Covid, Flu, RSV, whatever…Mainstream Medicine 'Explains' the Current Non-Covid Wave of Hospitalisations

Bonus features incl. a look at yet another junk 'study' from Switzerland and a couple of Nobel Prize nominations, that is, if I'd be asked

Today’s posting is a veritable gem of logic, reason, and sound argumentation—just kidding. Below you’ll find a piece that went live on Boxing Day (26 Dec.), published by the tabloid Heute, which seeks to explain why anyone mustn’t suspect anything about those damn modRNA injections. Speaking of the latter, see if you can spot the term ‘vaccine’ or its derivatives anywhere in the piece.

As always, the translation and emphases are mine, and so are the bottom lines.

Corona, Flu, and RSV Infections

In Austria, many people are currently suffering from respiratory infections. Some talk of an ‘immune debt’.

It has been hard to ignore for a few weeks now: people are coughing and sniffling everywhere [first time ever this happened, eh?]. Appointments are cancelled, children are taken out of school, and people at work are calling in sick. In some cases, Corona is the reason, but other viruses are also in the mix now. The flu viruses influenza A and B, as well as the respiratory syncytial virus (RSV) are also at the forefront. This is shown by overrun doctors’ offices and hospitals, as well as at wastewater surveillance data.

Some people attribute the accumulation of infections to the past Corona measures: because we kept our distance from one another for about two and a half years, wore masks, and avoided the pathogens as much as possible, our immune system was not trained. That is why it is now particularly vulnerable. They call this ‘immune debt’, or ‘immunodeficiency’ (see below). But the idea that we need constant infections and contact with viruses because otherwise the immune system shuts down is wrong.

No real evidence for ‘immunodedeficiency’

The term ‘immunodeficiency’ is still quite young. It was only invented last year, biochemist Emanuel Wyler, who researches the molecular biological processes in coronavirus infections at the Max Delbrück Center in Berlin, told the Tagesspiegel: ‘There is no real evidence for it so far, rather the term serves as a fighting term for those who see containment measures as a main cause of all evil in the pandemic.’

‘Our immune system is not a muscle’

‘The idea that you have to train the immune system like you do sports does not do justice to its complexity. We have no evidence that you have to be sick regularly to be particularly healthy’, Geneva-based virologist Isabella Eckerle told the science magazine Quarks.

‘The immune system is not a muscle that wastes, if I don’t exercise it’, seconds Carsten Watzl, Secretary General of the German Society for Immunology. It remains functional even [sic] without infections. After all, it is trained daily in dealing with countless microbes to which people are constantly exposed. Even [lol] in the last two and a half years, no one has lived in a sterile bubble. For Watzl, one thing is certain: ‘Sometimes it is also good not to go through an infection.’ [that guy is a genius; he should receive a Nobel Prize]

‘Immune gap’ instead of ‘immunodeficiency’

That’s how Carlo Cervia sees it, too: ‘You feel better without infections [strong contender for Watzl], if you stay healthy, your immune system doesn’t take any damage’, the immunologist from the University Hospital Zurich tells the Tagesanzeiger. He sees the fact that so many people are nevertheless currently plagued by respiratory illnesses rather explained by the lack of specific immunity against the usual pathogens that circulate every year: ‘Many are now getting sick because the immune memory for numerous pathogens is no longer completely up to date.’

The reason? The Corona measures were effective—against Corona as well as other viruses. That is why fewer people have become infected with respiratory pathogens in recent years. Thus, not only have the corresponding infections failed to materialise, but also the associated immune system updates (see below). Accordingly, the term ‘immune gap’ is better, says Cervia.

‘Immune system still works after Covid-19’

That the increase in respiratory infections this year has something to do with past Corona infections, as some people fear, is unlikely, according to Cervia. In a study, the immunologist and his colleagues found no evidence for this. In this study, the team had examined the functionality of T cells in 173 people after mild and severe Covid and found that there were no long-term abnormalities in the vast majority of patients, as the Tagesanzeiger reports. Cervia sees this as ‘good news’. According to this, the immune memory also functions after Covid-19.

However, the team still observed a slightly increased general activation of the T cells twelve months after the infection. ‘We don’t know the reasons for this. Possibly the immune system remains active because there is still inflammation somewhere’, says Cervia. It is unlikely that this long-lasting activation makes those affected more susceptible to other infections [perhaps the S protein is responsible?].

Immune System Updates

Viruses are the triggers of the respiratory infections currently circulating. And they are constantly changing—just like the Sars-CoV-2 coronavirus [that’s quite a hypothesis]. The new variants can multiply in the nose and throat [but not in your deltoid muscle after injection, I suppose]. The defence cells of our immune system then begin to fight the virus and the memory cells then remember the new variant [which is why we’re still using the Wuhan variant modRNA juice, eh?]. This means that every time we are sick, the immune system gets a kind of update. Thanks to this, it can fight off the mutations well the next time [based on my circles, that’s only true for the unvaccinated, but I digress]. Since in recent years far fewer people have fallen ill with, for example, influenza, many people are currently receiving the updates from previous years at the same time. [an abject lesson in how to contradict oneself in one paragraph]

Cold Season Ideal for Viral Spread

The fact that respiratory infections have increased so much, especially in the last few weeks, is due to several factors that are typical for the colder season and that contribute to viruses being able to spread more easily. For example, the air humidity is lower, which means that viruses remain stable in the air longer and can survive. In addition, our mucous membranes are more dried out. ‘If we breathe in viruses, they have an easier time penetrating through the mucous membranes’, Watzl explains. The fact that people spend more time indoors in colder temperatures also plays a role. And last but not least: when many people are ill, the risk of becoming infected oneself also increases—simply because more pathogens are circulating.

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Bottom Lines

This was a painful read, in particular the many silly things and omissions that we now know about, incl. the notion of ‘tolerance’ to Spike.

A brief glance at the study cited by Dr. Cervia, written by Taeschler and colleagues on the subject of T-cell recovery (see here) suggests that while they quite carefully constructed their sample, the authors failed to meaningfully relate their findings to the study population’s injection status. Uptake of one or two doses of modRNA juice is mentioned frequently, but it would appear to me (not a medical doctor) that statements such as this one (p. 2478; references omitted, line break and emphases mine):

A subgroup of severe COVID-19 patients had persistently low CD8+ T-cell counts up to 12 months after acute infection. If the reduction predated the SARS- CoV-2 infection, a proportionally reduced CD8 + naive T-cell repertoire could partially account for a delay in building an efficient virus-specific T- cell response. Persistent CD8+ T-cell lymphopenia was strongly associated with male sex, advanced age, increased inflammation, and CD8+ T-cell exhaustion during acute COVID-19.

So, what did we learn? Covid affects males more than females (I love biological facts) and is harder on elderly people. Nothing new here.

Increased levels of exhausted CD8+ T cells have been previously associated with an aging immune system. We only included very few patients with samples available prior to SARS-CoV-2 infection, precluding definite conclusions. However, the two patients that subsequently developed severe disease in this subgroup both had peripheral CD8+ T-cell counts in the low range before COVID- 19.

See that? The study is so small that it is ‘precluding definite conclusions’, which isn’t what the brainiac Dr. Cervia told the newspapers. I thought you should know. Not only was the sample very small (n = 173), they authors ‘only included very few patients with samples available prior to Sars-Cov-2 infection’, which also means that there’s no baseline. In my world, I’ll call such a design garbage in, garbage out.

Thus, our data suggest that, at least in a subgroup of patients, a pre-existing deficiency in CD8+ T-cell immunity could be associated with the development of an inflammatory phenotype and with severe COVID-19. Alternatively, it is conceivable that the apoptosis observed in severe COVID-19 could lead to persistently low CD8+ T-cell counts in the peripheral blood of a subgroup of patients, although we did not observe an increased frequency of apoptotic cells in this specific subgroup.

So, based on these ‘observations’ (ahem), the tentative conclusion is that some people in the study population were already at risk of health problems before Covid; alternatively, it may well be that severe Covid-19 induced cell death (apoptosis) due to high levels of inflammation.

Too bad the authors, while conducting a ‘multivariate analysis’ (p. 2472, pointing to Table 2 and Supplementary Material S1), fail to account for injection differentials. This is even more laughable as these are mentioned in Table 1, reproduced below:

I suppose the one thing we can learn here is that, according to the right-most highlighted section, 2/3 of ‘vaccinated’ ended up with a ‘breakthrough’ infection, which should putting questions pertaining to ‘vaccine efficacy’ to rest.

So, whatever, I suppose. I maintain that taking these modRNA juices is a kind of IQ test, which ‘the experts’ cited in the above pieces all fail to pass.

Just another day at the office, eh?